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Received an RCN/UBE grant from the National Science Foundation (subaward from Appalachian State University) for “Connectomes for Undergraduate Neuroscience Education and Learning (CUNEL).” The project is for three years. (2022)

Botero, V. [MHC '18], Stahl, B., Grenci, E.C., Boto, T., Park, S.J., King, L.B., Murphy, K.R., Colodner, K.J., Walker, J.A., Keene, A.C., Ja, W.W., Tomchik, S.M. (2021) Neurofibromin regulates metabolic rate via neuronal mechanisms in Drosophila. Nature Communications, Jul 13;12(1):4285. doi: 10.1038/s41467-021-24505-x.

Nangia, V. [MHC '16], O’Connell, J. ['19], Chopra, K ['20], Qing, Y.['21], Reppert, C. ['18], Chai, C. ['14], Bhasiin, K. ['22], and Colodner, K.J. (2021) Genetic reduction of tyramine β hydroxylase suppresses Tau toxicity in a Drosophila model of tauopathy. Neuroscience Letters, Apr 25: 135937. doi: 10.1016/j.neulet.2021.135937

Scarpelli EM, Trinh VY [MHC '18], Tashnim Z [MHC '19], Krans JL, Keller LC, Colodner KJ. (2019). Developmental expression of human tau in Drosophila melanogaster glial cells induces motor deficits and disrupts maintenance of PNS axonal integrity, without affecting synapse formation. PLoS One, 14(12):e0226380.

Shi Q.*, Colodner K.J.*, Matousek S.B., Merry K., Hong S., Kenison J.E., Frost J.L., Le K.K., Li S., Dodart, J.C., Caldarone B.J., Stevens B., Lemere C.A. (2015) Complement C3-deficient mice fail to display age-related hippocampal decline. Journal of Neuroscience, 35(38):13029-42.
* Co-first author.

Wang L, Colodner K.J., Feany M.B. (2011) Protein misfolding and oxidative stress promote glial-mediated neurodegeneration in an Alexander disease model. Journal of Neuroscience, 31(8): 2868 –2877. [Featured article]

Colodner K.J. and Feany M.B. (2010) Glial fibrillary tangles and JAK/STAT-mediated glial and neuronal cell death in a Drosophila model of glial tauopathy. Journal of Neuroscience, 30(48): 16102-13.